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Parkinson’s Basis to carry annual Transferring Day SF – NBC Bay Space

Daily SF News — Thu, 11 Jul 2024 02:24:33 +0000

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Speier to carry 1st summit on baby poverty in South San Francisco | Native Information

Daily SF News — Mon, 11 Sep 2023 20:30:47 +0000

Local leaders will gather in South San Francisco next week to discuss ending child poverty as part of the first summit held by the Jackie Speier Foundation.

From 8 a.m. to 4 p.m. Friday, Aug. 18, San Mateo County leaders will participate in a summit titled “San Mateo County Rising: Ending Child Poverty,” at the South San Francisco Conference Center. The event is being hosted by the Jackie Speier Foundation, a new organization started by former U.S. Rep. Jackie Speier to address the poverty gap for women and children.

“I’m excited about the opportunity to do what has not been done before — guarantee that no child in our county goes to bed hungry, lives unsheltered in a car, or suffers from physical and emotional violence or trauma,” Speier said in a press release.

Kicking off the summit will be a keynote address by Dr. Matthew Desmond, a professor of sociology at Princeton University and the Pulitzer Prize-winning author of “Evicted” and “Poverty, by America.” Emmy award-winning journalist Luz Peña will be the event’s master of ceremonies and opening addresses will also be given by Speier and Kathy Kwan, president of the Eustace-Kwan Family Foundation.

U.S. Rep. Kevin Mullin, D-South San Francisco; U.S. Rep. Anna Eshoo, D-Palo Alto; and county supervisors Ray Mueller and Noelia Corzo will also be featured speakers of the event with Mueller, Corzo and Speier expected to make a special initiative announcement.

The summit will also feature panel discussions on guaranteed income programs, child poverty and brain development and the effects of evictions on poverty before participants split into breakout groups to further explore eviction policies, youth mental health, local solutions to poverty and understanding a baby’s first year of life.

Additional speakers for those events will include Jim Higa, executive director of Philanthropic Ventures Foundation; Jim Pugh, co-director of the Universal Income Project; Dr. Greg Duncan, a professor at the University of California, Irvine; Dr. Kimberly Noble, professor of neuroscience and education at Columbia University; Dr. Lisa Chamberlain, a professor of pediatrics at the Stanford School of Medicine; Louise Rogers, San Mateo County chief of Health; Belinda Hernandez Arriaga, executive director of the nonprofit ALAS; Foster City Police Chief Tracy Avelar; County Attorney John Nibbelin; former Millbrae Mayor and Realtor Anne Oliva; and Chad Bojorquez, chief program officer of Destination Home.

State Sen. Josh Becker, D-Menlo Park; Redwood City Councilmember Alicia Aguirre; county Superintendent Nancy Magee; County Executive Officer Mike Callagy; Kitty Lopez, executive director of First Five San Mateo County; and Lenny Mendonca, the state’s former chief economic and business advisor, will lead breakout group discussions.

Co-sponsors of the event include the Chan Zuckerberg Initiative, the County of San Mateo, the Eustace-Kwan Family Foundation, Kaiser Permanente, Philanthropic Ventures Foundation and the San Mateo County Office of Education.

“It is unfathomable that in a county that is home to 22 billionaires and more than 5,000 people making over $4 million a year, 27,000 children live in poverty,” Speier said in the press release. “We will explore exciting strategies to raise children with the skills to fully thrive in our society.”

Visit jackiespeierfoundation.org/ for more information on the Jackie Speier Foundation and this month’s summit, “San Mateo County Rising: Ending Child Poverty.”

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San Francisco, Oakland marchers maintain mock funerals for public transit

Daily SF News — Thu, 08 Jun 2023 18:44:18 +0000

Considering that each procession was attended by pallbearers carrying flower-strewn coffins, the public transit funeral marches held in San Francisco and Oakland on Saturday had a decidedly celebratory atmosphere.

A brass quartet belted out “Amazing Grace” on Market Street. Oakland protesters energetically shouted “Gavin Newsom!” and responded. “Finance the bus!” The march in San Francisco was led by a man wearing a black suit and a long top hat.

“The call went out that we needed a little street theater and I said ‘sure,'” said Josh Kelly of San Francisco, who describes himself as a “safe roads and transit justice organizer” since my kids are small and I participate she had to go to school via Geary (boulevard).”

What drew Kelly, wearing the top hat, and more than 200 public transit advocates to rallies on both sides of the bay was a desire to increase Sacramento’s chances of receiving a $5 billion grant in the upcoming federal budget for local public transport. That money, spread over five years, would help cover operating deficits for systems like BART, Muni and AC Transit.

Public transit advocates march near City Hall during a fake funeral for public transit in San Francisco on Saturday, June 3, 2023. Video: Benjamin Fanjoy special on The Chronicle

Without them, operators and advocates warn, systems will have to cancel some routes and cut service on others – undermining progress in increasing ridership on buses and trains that have been paralyzed by the COVID-19 pandemic in 2020.

The state budget for 2023-2024 must be approved this month. Governor Gavin Newsom does not include a public transportation bailout in his proposed budget, citing the need to close a government funding gap estimated at $31 billion.

This short-term step would have long-term effects, speakers warned on Saturday.

“It’s so much easier to keep what we have than to restore it later,” said Jaime Viloria, a Tenderloin resident who was a speaker at the rally at Civic Center Plaza that followed the march down Market Street introduced. “We need the bridge funding” to maintain current service levels until a long-term solution is found, such as a regional transit tax.

A pro-transit group marches on Market Street in San Francisco for a rally and simulated pro-transit funeral procession.

Benjamin Fanjoy/The Chronicle Special

The idea of a mock burial came up last week and gives the individual marches an adrenaline rush. In San Francisco, for example, there were calls for volunteer pallbearers before a crowd of about 125 people made their way from United Nations Plaza, with Kelly impromptu preaching a sermon of supposed commemoration.

“The cause of death is neglect,” he stressed. “The cause of death is abandonment. … Gov. Gavin Newsom is ready to pull the plug.”

The closest a Newsom defense attorney came to was state senator Scott Wiener, who helped drive the push for a virtual five-year extension of state transit subsidies, an early response to the pandemic.

“There’s a lot of good in the governor’s budget — it preserves so much of the safety net,” Wiener told the crowd, which had grown to around 200 at the time of the rally outside City Hall. “But in this area the budget is simply not enough.”

Brandon Yung helps carry a model AC Transit bus and replica coffin at the Transit rally in downtown San Francisco on Saturday.

Benjamin Fanjoy/The Chronicle Special

Speakers emphasized the role of transit in reducing carbon emissions and its importance for marginalized groups in society, such as low-income, elderly or disabled residents. Frustration over conditions with systems like BART has been little spoken of – a factor cited by former and current drivers in a recent survey.

“It’s hard to imagine San Francisco and the Bay Area without excellent public transportation,” Wiener said. “We should never take it for granted.”

At the same time, Wiener said that talks with other lawmakers combined with pressure from public transport fans could create an opportunity for some sort of subsidy in the upcoming budget: “I’m more optimistic than ever that we’ll get this resolved,” so additional cuts are not necessary.

In Oakland, speakers were AC Transit Directors Jean Walsh and Jovanka Beckles, and BART Director Lateefah Simon. Public transport advocates taking the mic at the San Francisco rally included Mayor London Breed and supervisors Rafael Mandelman and Dean Preston.

A group carries a model Caltrain car during a mock funeral procession in San Francisco, a rally for public transit funding. Proponents called for more funding to be allocated to the state’s public transit companies.

Benjamin Fanjoy/The Chronicle Special

“These are a merry bunch for a funeral procession,” Preston remarked at the beginning of his speech.

The idea of mock catulls for a mock funeral “came naturally out of the idea of a (financial) death spiral,” said Lian Chang, board member of Walk San Francisco. She also collected the playful protest art during three hectic nights with friends at her Richmond District apartment.

However ad hoc the result, “I’m glad we made it,” Chang said. “I’m not happy with the circumstances, but it was a way to draw attention to the seriousness of the (possible) cuts if no solution is found over the next few days.”

Reach John King: jking@sfchronicle.com; Twitter: @johnkingsfchron

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Advocates Maintain Mock Funeral, Rally for Public Transit in San Francisco – NBC Bay Space

Daily SF News — Sun, 04 Jun 2023 14:18:07 +0000

Public transit advocates rallied in San Francisco Saturday to support Bay Area public transit with a different kind of march: a mock funeral and rally.

The crowd lamented the potential loss of public transport and the impact on the local economy if the state didn’t provide urgent funds, they said.

“Without this bridging funding, just to get through the difficult period when ridership has not yet returned, agencies will have to stop their services,” said Lian Chang, a public transport advocate.

The group marched, stating that transit companies have kept essential workers moving during the pandemic, but federal aid is running out and they want the state to step in.

“So we need a little more funding, firstly to continue the recovery and secondly to get to where we can adopt a regional measure, a local measure, to keep the bus running, to keep the train running,” he said Cyrus, supporter of public transport hall.

As the rally grew, so did the crowd near San Francisco City Hall.

State Senator Scott Wiener addressed the crowd.

“We know how to solve this. “We need about $1 billion a year for five years, which is totally doable in the state budget, and we’re going to keep fighting over the last week to make that happen,” he said.

Wiener shared his perspective on what is happening in Sacramento.

“I am now more optimistic than ever that we will solve the problem,” he said. “Both houses of the legislature have said they want to resolve the issue and we now need to work it out with the governor.”

Advocates said they would continue to advocate for those who rely most on public transport.

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Viruses Hiding Out in Sufferers Maintain Lengthy Covid Reply

Daily SF News — Tue, 14 Mar 2023 09:03:29 +0000

Mutations showed the virus was “running like hell.”

Tracking microbes through a foul-smelling network of sewers led virologist Marc Johnson to the source of unusual coronavirus mutants.

Although Johnson could not identify this individual, he was able to use genetic data to see that virus particles had been freshly made and expelled for more than a year – many times longer than a typical two-week Covid infection.

And during that time, the mutations showed the virus was “running like hell,” trying to evade the person’s immune system, Johnson said. Laboratory analysis of his sewage samples revealed the battlefield in the patient’s body where the virus was rapidly evolving to claim a stronghold.

“You can find a lot of chronic infections — people who have probably been infected for over a year — where the virus hasn’t changed at all,” he said. “And I don’t understand why it just goes crazy in some patients.”

As the scourge of Covid-19 enters its fourth year, patients like the one Johnson discovered by being traced through miles of drainpipes and drains are bringing researchers closer to answers to key questions: Where do worrying new mutants come from? And what role do they play in Long Covid, the mysterious post-infection disease that affects more than 140 million people worldwide?

Scientists are studying the possibility that some of the most contagious versions of the coronavirus — Omicron and its descendants — came from chronically infected individuals whose immune systems were weakened by illness, drugs, or both. Research published in December shows the virus can persist in the body and brain for months. That suggests it may be hiding in human cells and tissues, similar to HIV and the chickenpox virus that causes shingles.

Fascinating autopsies

Traces in the blood and stool of patients with long-lasting symptoms indicate that SARS-CoV-2 could be stored in the intestines, fat or other tissues that offer protection against the body’s immune system. Researchers from the US National Institutes of Health, who conducted painstaking autopsies on the bodies of 44 Covid victims, found viral genetic material in the bodies and brains of the patients up to 7 1/2 months after symptoms began. In one case, virus particles isolated from the brain were grown in a laboratory dish, proving that they were fully functional and capable of replication.

“The predominant damage still seems to be in the lungs,” said Daniel Chertow, who led the research in the NIH’s Emerging Pathogens Division, “but oh man, we really need to better understand what kind of damage is happening with all these others.” is done sets.”

Most of those autopsied were elderly and ill before contracting Covid, and all died before vaccines became available. And while no one was known to have had Covid for long, the findings, published in the journal Nature, still require follow-up.

“It is imperative that we try to understand in great detail what role persistence of viral RNA and other viral components might play in long-Covid,” Chertow said.

No one knows if the coronavirus or its remnants are still present in everyone who has had Covid, or if it’s just a group of patients, said Timothy Henrich, an associate professor of medicine at the University of California, San Francisco.

“We all hypothesize that it’s a driver of Long Covid, but we really haven’t shown that definitively,” Henrich said. “This is still something that needs to be done.”

Still, Chertow’s research has already inspired the experimental use of antiviral drugs like Pfizer Inc.’s Paxlovid to see if it can eradicate viral reservoirs and relieve long-term symptoms. Even a relatively small number of infected cells could trick the immune system into causing inflammation, blood clots, and other problems associated with long Covid, according to Amy Proal, co-founder of the PolyBio Research Foundation, a Boston-based nonprofit that facilitates research into chronic, infection-associated conditions .

It’s “the most logical explanation because it pretty much explains everything else,” she said.

Scan hideouts

Henrich, who has helped develop sophisticated imaging techniques capable of locating HIV in tissues, plans to use the same approach to find Covid’s hiding places. Dozens of patients will undergo a series of full-body scans looking for signals of viral protein production or persistence. These will be compared to symptoms to see how the continued presence of the coronavirus correlates to a long covid.

The group has started scanning patients to look for protective T cells that could indicate an aberrant immune response to SARS-CoV-2. They examine biopsies of the participants’ digestive tracts for traces of the virus, Henrich said.

A particular focus is on lymphoid tissues, which produce, store and transport T cells that fight infection, and antibody-producing B cells. The coronavirus could also be hiding in long-lived nerve cells and heart muscles, where it can bring on chest pain, brain fog, fatigue and other long-standing Covid symptoms, said Diane Griffin, a virologist at the Johns Hopkins Bloomberg School of Public Health in Baltimore who has been studying for more than 50 years studying the body’s response to viral infections.

“We have pretty good information from acute RNA virus infections that RNA persists and that it has consequences,” said Griffin, who is vice president of the US National Academy of Sciences. “Basically, it’s difficult to get rid of viruses.”

Proving that prolonged viral infection causes long-term Covid will be difficult, Griffin said. Viruses in stealth mode suppress replication so as not to damage their host cells. Despite the extensive distribution of coronavirus RNA in the patients’ bodies, Chertow’s team saw little evidence of inflammation or that the immune system had attempted to destroy infected cells outside of the airways.

Viral Evolution

Interestingly, when the NIH scientists analyzed the genetic makeup of virus samples collected from six patients, they found versions in the lungs that differed from those collected in other tissues. In one patient, the viruses found in two brain regions – the thalamus and the hypothalamus – were significantly different, suggesting that certain mutations favored the pathogen’s persistence there.

“We know that these RNA viruses have the potential to evolve within a host,” Chertow said. “The more they can replicate and the longer the time they have to do so and the higher the level of replication that takes place, the more opportunity these viruses have to evolve.”

This raises an interesting possibility: as the virus evolves to inhabit different organs and tissues, the process can produce more and more infectious and immune-evasive variants. Nobody knows yet whether this is the case, but Johnson’s wastewater analysis at the University of Missouri gives the first indications.

Out of control pathogens

Since the early days of the Covid pandemic, researchers have known that Covid is capable of infecting the digestive tract, causing stomach upset and causing patients to shed traces of the virus in their faeces. Johnson routinely scans around 100 sewer networks in the Midwest for unusual SARS-CoV-2 strains, called cryptic lineages because their source is unknown.

“They’re rare, but they’re out there,” he says.

When Johnson started work in March 2021, he had no idea where the journey was going. Then, after more than a year ago, omicron drove US Covid cases to a record high, he began actively looking for strains with pronounced genetic changes.

He discovered one that came from a drain in Wisconsin that served 100,000 people. Its genetic signature was much more different than previous versions of Omicron, but it had not been reported in any patient. Importantly, its mutations appeared in newer versions of omicron. It was almost as if Johnson saw where the virus was going — evolutionarily speaking — before it arrived.

“So we started saying, let’s find out where it’s coming from,” he said.

Black Swan

For four months he and his colleagues went from one shaft to the next in an unknown metropolitan area. Then they discovered that the samples came from a building. Then one side of the building. Then half a dozen toilets, regularly used by about 30 people.

There, the investigation had come to a standstill, said Johnson. Despite this, he was able to deduce that the cryptic lineage came from an individual who was infected nearly two years ago, as it evolved from a strain last discovered in Wisconsin in April 2021.

In August, the concentration of the variant was 1.5 billion copies per liter of wastewater. For comparison, at the height of a massive Covid outbreak in a Missouri prison, a liter of sewage contained just 100 million copies of the coronavirus.

“It’s insane how much virus this person is shedding,” Johnson said.

In December, the same variant appeared in the toilet installation in Wisconsin for the 13th consecutive month.

“Whoever this person is, they still seem to be going to work every day and have been for a while,” Johnson said. “It seems they don’t know they are infected. It can’t be good for her.”

The line hasn’t appeared anywhere else, suggesting it’s not spreading, Johnson said. But there’s no question that it can grow, as evidenced by long-standing high concentrations in toilet drains.

“The question is why?” he said. “We assume something like this happened with omicron and that there was some sort of black swan event that allowed the virus to get out.”

Johnson says studying cryptic lineages challenged his thinking about the coronavirus’ ability to lodge itself in human tissues, particularly outside the respiratory system, and its potential to cause prolonged illness.

“I’m particularly open now to the idea that there are secondary infections that we just don’t know about,” he said. “And maybe that helps explain some of these very weird long Covid symptoms.”

(Except for the headline, this story was not edited by NDTV staff and was published by a syndicated feed.)

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Viruses hiding out in sufferers maintain solutions to lengthy COVID | Nationwide

Daily SF News — Sun, 05 Mar 2023 18:30:15 +0000

Tracking microbes through a foul-smelling network of sewers led virologist Marc Johnson to the source of unusual coronavirus mutants.

After months of sampling sewage, the microbiologist from the University of Missouri School of Medicine found out exactly where the mutants came from: from a regular user of the toilets at a certain Wisconsin company. Although Johnson could not identify this individual, he was able to use genetic data to see that virus particles were freshly made and expelled for more than a year – many times longer than a typical two-week COVID-19 infection.

As the scourge of COVID-19 enters its fourth year, patients like the one Johnson discovered when traced down miles of drainpipes and drains are bringing researchers closer to answers to key questions: Where do worrying new mutants come from? And what role do they play in Long COVID, the mysterious post-infection disease that is affecting more than 140 million people worldwide?

Scientists are studying the possibility that some of the most contagious versions of the coronavirus — Omicron and its descendants — came from chronically infected individuals whose immune systems were weakened by disease, drugs, or both. Research published in December shows the virus can persist in the body and brain for months. That suggests it may be hiding in human cells and tissues, similar to HIV and the chickenpox virus that causes shingles.

Exciting autopsies

Traces in the blood and stool of patients with long-lasting symptoms indicate that SARS-CoV-2 could be stored in the intestines, fat or other tissues that offer protection against the body’s immune system. Researchers from the US National Institutes of Health, who performed painstaking autopsies on the bodies of 44 COVID-19 victims, found viral genetic material in the bodies and brains of the patients up to 7 1/2 months after symptoms began. In one case, virus particles isolated from the brain were grown in a laboratory dish, proving that they were fully functional and capable of replication.

“The predominant damage still seems to be in the lungs,” said Daniel Chertow, who led the research in the NIH’s Emerging Pathogens Division, “but, oh boy, we really need to better understand what kind of damage is in all of these.” other places are prepared.”

Most of those autopsied were elderly and ill before contracting COVID-19, and all died before vaccines became available. And while no one was known to have had COVID for long, the findings, published in the journal Nature, still call for follow-up.

“It is imperative that we try to understand in great detail what role persistence of viral RNA and other viral components might play in long COVID,” Chertow said.

No one knows if the coronavirus or its remnants are still present in everyone who has had COVID-19, or if it’s just a group of patients, said Timothy Henrich, an associate professor of medicine at the University of California, San Francisco .

“We all hypothesize that it’s a driver for long COVID, but we really haven’t shown that definitively,” Henrich said. “This is still something that needs to be done.”

Still, Chertow’s research has already inspired the experimental use of antiviral drugs like Pfizer Inc.’s Paxlovid to see if it can eradicate viral reservoirs and relieve long-term symptoms. According to Amy Proal, co-founder of the PolyBio Research Foundation, a Boston-based nonprofit dedicated to research into chronic, infection-associated conditions.

It’s “the most logical explanation because it pretty much explains everything else,” she said.

Scan hideouts

Henrich, who has helped develop sophisticated imaging techniques that can pinpoint HIV in tissues, plans to use the same approach to find COVID-19’s hiding spots. Dozens of patients will undergo a series of full-body scans looking for signals of viral protein production or persistence. These will be compared to symptoms to see how the continued presence of the coronavirus correlates to a long COVID.

A particular focus is on lymphoid tissues, which produce, store and transport T cells that fight infection, and antibody-producing B cells. The coronavirus could also be hiding in long-lived nerve cells and heart muscles, where it can cause chest pain, brain fog, fatigue and other long-lasting COVID symptoms, said Diane Griffin, a virologist at the Johns Hopkins Bloomberg School of Public Health in Baltimore, who has been studying for more than 50 years studying the body’s response to viral infections.

Proving that prolonged viral infection causes long-term COVID will be difficult, Griffin said. Viruses in stealth mode suppress replication so as not to damage their host cells. Despite the extensive distribution of coronavirus RNA in the patients’ bodies, Chertow’s team saw little evidence of inflammation or that the immune system had attempted to destroy infected cells outside of the airways.

Viral Evolution

“We know that these RNA viruses have the potential to evolve within a host,” Chertow said. “The more they can replicate and the longer they have time to do so and the higher the level of replication that takes place, the more opportunities these viruses have to evolve.”

Out of control pathogens

Since the early days of the COVID-19 pandemic, researchers have known that the disease can infect the digestive tract, causing stomach upset and causing patients to shed trace amounts of the virus in their feces. Johnson routinely scans around 100 sewer networks in the Midwest for unusual SARS-CoV-2 strains, called cryptic lineages because their source is unknown.

“They are rare, but they exist,” he says.

When Johnson started work in March 2021, he had no idea where the journey was going. Then, after more than a year ago, omicron drove US COVID-19 cases to a record high, he began actively looking for strains with pronounced genetic changes.

“So we started saying, let’s find out where it’s coming from,” he said.

Black Swan

In August, the concentration of the variant was 1.5 billion copies per liter of wastewater. For comparison, at the height of a massive COVID-19 outbreak in a Missouri prison, a liter of sewage contained just 100 million copies of the coronavirus.

“It’s insane how much virus this person is shedding,” Johnson said.

In December, the same variant appeared in the toilet installation in Wisconsin for the 13th consecutive month.

“Whoever this person is, they still seem to be going to work every day and have been for a while,” Johnson said. “It seems they don’t know they are infected. It can’t be good for her.”

“The question is why?” he said. “We assume something like this happened at omicron and that there was some sort of black swan event that allowed the virus to get out.”

Johnson says studying cryptic lineages has challenged his thinking about the coronavirus’ ability to lodge itself in human tissues, particularly outside the respiratory system, and its potential to cause prolonged illness.

“I’m particularly open now to the idea that there are secondary infections that we just don’t know about,” he said. “And maybe that helps explain some of these very oddly long COVID symptoms.”

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Pamplin Media Group – Diamond Beat: Seattle’s Margevicius, relievers maintain Houston to at least one hit

Sat, 15 May 2021 09:51:44 +0000

12-18 April: Daily results log for Area Pro, college and amateur baseball and softball teams.

Diamond Beat is the place to find results and updates on baseball and softball action in the area, including coverage of the Seattle Mariners, as well as college baseball and softball.

SUNDAY APRIL 18th

Pro baseball

Mariners 7, Astros 2 – Seattle (10-6) won the three-game series with Houston at T-Mobile Park. The Astros only managed one goal from starter Nick Margevicius in the second inning. Margevicius threw four innings and five relievers threw a no-hit ball. Ty France hit a homer with two runs and Mitch Haniger had a double and triple, as well as three RBIs and two runs.

Seattle will play the Los Angeles Dodgers at home for the next two nights.

College baseball

Pilots 8, Lions 2 – Portland (18-17, 10-5) won the three-game series with a visit to Loyola Marymount. Chad Stevens and Hunter Montgomery met UP; Stevens had scored three hits, two RBIs and three runs. Sam Brown went 4 on 4.

The pilots play in a series of three games on Tuesday at OSU and next weekend in San Francisco.

Beavers 5, Bears 3 – Oregon State (24-10, 10-5 Pac-12) won the three-game series in Cal. Ryan Ober and Garret Forrester each drove in two races. Dylan Beavers met Cal.

The Beavers will host Portland on Tuesday and three games at UC Irvine this weekend.

Ducks 7, Trojans 5 – Oregon (22-8, 8-4 Pac-12) scored four runs in the eighth inning and won the three-game series at USC. Sam Novitske and Gabe Matthews had three hits each; Novitske drove in three runs, Matthews scored three runs.

The ducks are hosting UCLA for three games next.

College softball

Bruins 6, Beavers 0 – Oregon State (15-20, 3-12 Pac-12) only managed two hits. In the three games at UCLA, the Beavers had three hits and were beaten 20-0. Maya Brady returned to UCLA. After 12 days, the Beavers will play four games at Stanford starting April 30th.

Huskies 6, Ducks 1 – Oregon (28-8, 6-6 Pac-12) lost three of four games in Seattle. UWs Gabbie Plain took the lead 23-0. Morganne Flores and Sami Reynolds each faced UW.

Oregon went 3-1 against both UCLA and Washington in the past two weeks. The Ducks will host four games at Stanford this weekend.

SATURDAY 17TH APRIL

Pro baseball

Astros 1, Mariners 0 – Zack Greinke allowed four hits in eight innings, and Ryan Pressly ended when Houston beat Seattle at T-Mobile Park. Taylor Jones’ RBI single in the fourth inning overshadowed the only run of the game. Seattle starter Chris Flexen gave up 10 hits and one run in six innings.

College baseball

Ducks 13, Trojans 4 – Oregon scored four home runs from Anthony Hall, Kenyon Yovan, Jack Scanlon and Gavin Grant; Scanlon and Hall hit 3-run shots. Gabe Matthews, now UO’s all-time leader in hits, had three hits and two RBIs.

Beavers 2, Bears 1 – Ryan Obers single ran in the tie run and then Jacob met Melton on the throw of the game when the State of Oregon caught up in the sixth inning. Cooper Hjerpe allowed three hits in sixth innings, and Jake Mulholland got the save and set the School save record (38, previous Kevin Gunderson record, 37 from 2004-06).

Pilots 9, Lions 1 – Ben Patacsil (two-run shot) and Sam Brown each arrived when Portland defeated Loyola Marymount. Bradley McVay improved to 5-1 on the hill and Jacob Dobmeier allowed a hit in three innings of relief.

College softball

Huskies win 4-2, Ducks 10-1 – At the start, Sami Reynolds from Washington met and Gabbie Plain threw himself to 22-0. Oregon’s Shaye Bowden came home. In the second game, Oregon beat UW in a five-inning game when Rachel Cid returned home and ran four RBis and avenue bunkers in three runs.

Vikings 8, Bengals 4 – Paetynn Lopez hit a Grand Slam home run for Portland State (7-22, 4-8 Big Sky) versus Idaho State.

Bruins 7, Beavers 0 – Megan Faraimo allowed a hit and knocked out 17. The second game of the double header has been canceled.

FRIDAY APRIL 16

Pro baseball

Mariners 6, Astros 5 – Houston led 5-2, but Seattle rallied to win. Ty France was awarded the winning run in the ninth inning at JP Crawford. Evan White had finished the game with a homer from the eighth inning.

College baseball

Trojans 9, Ducks 5 – Tyresse Turner hit a two-run homer in the seventh inning and Ben Ramirez hit a two-run double in the eighth inning as USC rallied against Oregon. Ramirez also tripled in the run.

Oregon State 15, Cal 8 – Jacob Melton had four hits, including a triple and a home run, and six RBIs. Ryan Ober hit a grand slam. The beavers had 19 hits.

Lions 8, Pilots 5 – Loyola Marymount had 16 hits. Briley Knight returned to Portland.

College softball

Huskies 8, Ducks 0 – Oregon lost their fourth game in a row. The ducks managed just two hits ahead of Gabbie Plain (21-0), who scored the 12th goal. Baylee Klinger and Sami Reynolds met UW.

Bruins 7, Beavers 0 – Rachel Garcia threw a no-hitter with 12 strikeouts against the State of Oregon. The Bruins had 14 hits, including a homer from Aaliyah Jordan.

Pilots win 8-0, Bengals win 3-2 – Portland State and Idaho State share a double header. Oiivia Gray from the PSU only allowed two hits in the opener. Angelica Cano faced ISU in the second game.

THURSDAY APRIL 15th

Pro baseball

Mariners win 4-2, 2-1 – Seattle went 5-2 on its road trip and culminated in a double header in Baltimore. Mitch Haniger scored in both games, including a two-run shot that connected the prelude. JP Crawford’s double with two runs in the sixth inning gave the Mariners the lead. Marco Gonzales allowed only three hits in the opener and a Trey Mancini homer with two runs in five innings. Dylan Moore and Haniger faced starter Justin Dunn in the second game, who allowed two hits in five innings.

The Mariners will play three games against Houston and then two against the Los Angeles Dodgers at T-Mobile Park starting Friday night.

WEDNESDAY APRIL 14th

College baseball

Ducks 18, Pilots 1 – Oregon attended the University of Portland and beat the pilots. Josh Kasevich went 5v6 with six RBIs and four runs scored. The ducks had 21 hits. Gabe Matthews had two hits, connecting JJ Altobelli (2010-13) to the all-time UO hit mark (211). Matthews has played in 200 games; Altobelli played 229.

TUESDAY APRIL 13th

Pro baseball

Mariners win 4-3, Orioles 7-6 – In a double header, the Mariners took the first game after Monday game postponed due to weather conditions when Ty France and Tom Murphy returned home and Kyle Seager scored an RBI double in the top of the eighth inning . Ramon Urias returned to the Orioles.

Then Urias overtook the winning run for Baltimore in the seventh inning of the second game. Maikal Franco had three RBIs for the Orioles. Jose Marmolejos (3-Run Homer) and Sam Haggerty (2 Run Run) each faced the Mariners.

College baseball

Huskies 6, Pilots 4 – In an out-of-league game at the University of Portland, the Pilots scored three runs in the ninth inning to close it but failed to break the deficit. Portland’s Ty Saunders had three hits, including a triple and a home run. UP plays Oregon at home at 2pm on Wednesday.

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